From Gastrulation to Cancer: Inflammation as the (Patho) Physiologic common soil and the regulatory role of Neural Therapy
Colombia 2026 – Applied Neurophysiology and the Study of Autonomic Nervous System Physiology and Pathophysiology
Dear friends and colleagues!
It is a great honor and pleasure for me to be invited to one of the most important Congresses in 2026 in the field of Applied Neurophysiology and the Study of Autonomic Nervous System Physiology and Pathophysiology. The Event will take place in Colombia which is one of the “Superpowers” of the therapeutic use of Local Anesthetics (Neural Therapy), a place where NT is officially being taught in the Medical School of the University of Bogota.
The Lecture which I will hold , deals with the very interesting and complicated phenomenon of Inflammation.
The title is: “From Gastrulation to Cancer: Inflammation as the (Patho) Physiologic common soil and the regulatory role of Neural Therapy”.
Some Information to grasp its importance:
Inflammation is not merely a reaction to injury—it is a primordial biological program embedded in life itself. Even during gastrulation, the earliest stage of embryonic development, cells express behaviors—migration, matrix remodeling, cytokine signaling—that mirror what we later recognize as inflammatory processes. Inflammation, therefore, is not inherently pathological; it is a fundamental organizing principle of biology.
Physiological inflammation is essential for defense, repair, and regeneration. The problem is not inflammation per se, but its failure to resolve. When resolution mechanisms break down, acute inflammation shifts into chronic low-grade inflammation—a state now recognized as a shared substrate of cardiovascular disease, metabolic disorders, neurodegeneration, depression, and cancer. Tumors, in particular, resemble wounds that do not resolve, reactivating embryologic programs such as angiogenesis and epithelial-to-mesenchymal transition.
At the center of this dysregulation lies the stress system. The neuro-immune network—integrating the HPA axis, sympathetic activity, and vagal tone—continuously shapes inflammatory setpoints. Chronic stress sustains pro-inflammatory signaling and destabilizes regulation.
In this context, simply suppressing inflammation is not equivalent to restoring health. What is required is regulation. Neural Therapy, through the targeted use of local anesthetics, aims to modulate autonomic imbalance, neurogenic inflammation, and dysfunctional reflex circuits. Rather than silencing mediators, it seeks to restore communication within biological networks.
From development to chronic disease, inflammation is a continuous thread. The challenge is not to eliminate it, but to regulate it—recognizing it as the common soil of both health and pathology.
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